Figure 4 Sample Clauses
Figure 4. Coordinates of the center of the two batteries ventilation holes (per battery pack). These coordinates are given with reference to the S/C coordinate system origin located at the center of the S/C separation plane (X, Y, Z) : A= 1042.425 , -857.5 , -145.75 B= 1042.425 , 857.5 , -145.75 D= -1042.425, -857.5, -145.75 C= -1042.425, 857.5, -145.75 Frequency (MHz) E_Field (dBmV/m) The susceptibility level is at equipment surface. To evaluate the effect of the launcher radiated field on the L/V onto S/C TC receivers, the launcher radiation level at S/C separation plane region must be attenuated by 33 dB to account for the presence of S/C structures and MLI and the location of the TC receivers inside the S/C. INMARSAT-4 radiated emissions (TM emitters OFF)
Figure 4. 3: (a) Observation and (b) schematic representation of the buildup of one fringe (m = 1) on the mirror under slow-scan imaging. The 6 bright spots (numbered) are a result of off-axis injection into a N = 6 degenerate cavity. The ellipses are formed by light that is scattered out of the six hit points into periodic orbits. Only the ellipses that interfere constructively after one round-trip (total path length equals λ ) are visible. The turning points of the scatter ellipses are observed as the fringe (dotted circle), which has a diameter of 1 cm.
Figure 4. Figure 5. Participants
Figure 4. 2 below shows the major existing developments at the surroundings of the Project Area within the Study Area. The heights (in mPD) of the developments are also listed. Project Area 1. Project Area (proposed 110mPD), currently Hong Kong School of Motoring (~9.6mPD) 2. Horizon Plaza (110.5mPD), Electric Tower (118.3mPD) Ap Xxx Xxxx Industrial Estate Station Building (27.6mPD)
Figure 4. 16-bit Stack Frame on 32-bit Stack
Figure 4. 2 Restriction, matching and exclusion process for selection of intervention and control villages (1), and timeline for study rounds and outcome data collection (2) 47 Figure 5.1 Profile of the study population across four rounds of data collection. The total number of individuals included at each stage of enrollment, follow-up and analysis are on the left in the intervention and control columns. The subset of the total population that is under 5 years is in right in dashed boxes 66 Figure 6.1 Environmental sample observations by sample type and assay across the four study rounds 94
Figure 4. 2.4 a) Schemes adopted in the numerical analyses performed by Xxxxxxx et al., 2008; b)
Figure 4. What kind of learning information do you wish to know about your children? (Score, localisation …)? Figure 5: What disabilities should Beaconing provide support for? Figure 6: What could be the main security issues?
Figure 4. At sacrifice, blood and spleen cells were isolated (n=5 per group) and percentages of CD4+ and CD8+ T cells were determined with flow cytometry (A). In addition, CD4+CD62Llow effector T cells and CD4+CD44low62Lhigh naive T cells were determined in the spleen (B). Splenic T cell subsets were determined by staining for CD4 and the transcription factors T-bet (Th1), GATA-3 (Th2), RORγt (Th17) B Naive Effector D and Foxp3 (CD25+ Tregs) (C). The effect of MDSC administration % within CD4+ T cells 15 60 on spleen cell proliferation was 9 50 S.I. T-bet GATA-3 ROR t + Foxp3 CD25 Control MDSCs determined by culturing splenocytes (n=5 per group) in the presence or absence of CD3/CD28 stimulation (D). Proliferation was assessed by the amount of 3H-thymidine incorporation in dividing cells. The proliferation is expressed as stimulation index. ***P<0.001 population of the spleen compared with control mice (Figure 4C). In contrast, Th2 and Treg responses remained unchanged following adoptive transfer of MDSCs. Although adoptive transfer of MDSCs decreased the pool of splenic effector CD4+ T cells and more specifically, decreased Th1 and Th17 cell subsets, we did not observe any difference in splenocyte proliferation of MDSC-treated mice (stimulation index of 49.7±3.6) in comparison with control mice (stimulation index of 51.8±2.0) after stimulation with αCD3/CD28 (Figure 4D). Adoptive transfer of MDSCs also affected B cell responses since MDSC-treated mice had reduced circulating CD19+ B cells (23.7±1.4% vs. 30.3±2.3% in control mice, P<0.05, Figure 5A). More particular, we observed a 30% decrease in circulating B2 cells in MDSC-treated mice (15.2±1.0%) in comparison with control mice (21.6±2.1%, P<0.05, Figure 5B). Furthermore, we found that splenic B cells of MDSC-treated mice proliferated less vigorously (29.5±1.4% CD19+Ki-67+ cells) than B cells of control mice (35.9±1.2% CD19+Ki-67+ cells, P<0.05, Figure 5C). We also determined oxLDL-specific antibodies in serum (Figure 5D) but did not find any difference in MDSC-treated mice in comparison with control mice. A % CD19+ cells C Control MDSCs % Ki-67+ within CD19+ cells % B2 cells OD 450 nm